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Type 2 diabetes mellitus T2DM is one of the leading causes of morbidity and mortality. While all ethnic groups are affected, the prevalence of T2DM in South Asians, both in their home countries and abroad, is extremely high and is continuing to rise rapidly.

Innate biological susceptibilities coupled with rapid changes in physical activity, diet, and other lifestyle behaviors are contributing factors propelling the increased burden of disease in this population. The large scope of this problem calls for investigations into the cause of increased susceptibility and preventative efforts at both the individual and population level that are aggressive, culturally sensitive, and start early.

In this review, we outline the biological and environmental factors that place South Asians at elevated risk for T2DM, compared with Caucasian and other ethnic groups. Type 2 diabetes mellitus T2DM currently affects approximately million people worldwide. Much of this heterogeneity can be attributed to differing states of socioeconomic development, variations in lifestyle factors, and differences in prevalence of undiagnosed versus diagnosed diabetes among countries.

The majority of T2DM data from South Asia have come from India, the South Asian country with the largest diabetes burden, and where the prevalence has increased steadily over the past 40 years. The overall weighted prevalence was Source: International Diabetes Federation. By it is projected that there will be The prevalence of T2DM is also high among South Asian migrant populations, with several studies noting a higher prevalence of T2DM in migrant South Asians than in other ethnic groups in the host countries.

These prevalence estimates greatly exceed those in non-Hispanic whites 7. The higher prevalence and incidence of T2DM in South Asians populations worldwide is likely indicative of underlying biological factors in South Asians, coupled with recent rapid changes in dietary, activity, and other lifestyle behaviors. Herein, we discuss the current literature comparing South Asians with other populations with regard to biological e.

A synopsis of the paper is presented in Table 2. Comparison of diabetes risk factors among South Asians, Caucasians, and other ethnic groups. A study by Mohan et al. It appears that some of the increased propensity for South Asians to develop insulin resistance could be attributable to greater accumulation of visceral fat.

A recent study comparing young South Asian and Caucasian women matched for BMI noted no differences in body fat distribution and insulin sensitivity between groups. Therefore, generalizations cannot be made about women of all ages and BMI categories, and further studies exclusive to South Asian women are warranted. A study comparing insulin secretion in 12 insulin-resistant adults who were born with intrauterine growth restriction IUGR to 13 controls adults failed to observe any evidence suggesting a defect in insulin secretion in young adults born with IUGR.

This could have severe implications for T2DM development in South Asians due to the high prevalence of low birth weight babies born in the region. However, the complete mechanistic pathway determining T2DM in this population remains to be elucidated and further studies are warranted. While some studies have noted associations between T2DM and FTO that were mediated by obesity, 49 others have reported associations with FTO variants that were not entirely mediated by BMI, central adiposity, or obesity.

However, there is also considerable heterogeneity as measured by associations among different South Asian populations, which calls for further large scale studies on South Asians of varying cultural, geographic, and economic backgrounds. Another possibility is that varying genetic polymorphisms could contribute to differences in T2DM risk.

Adiponectin is a protein associated with glucose modulation, 52 and studies have noted lower levels of circulating plasma adiponectin in South Asians, compared with Caucasians. It is a widely held belief that T2DM onset occurs at a much earlier age in South Asians than in other ethnic populations.

There is also a lack of information regarding differences in age-specific prevalence between South Asians living in South Asia and those who have migrated to other countries.

Such studies may be able to elucidate whether South Asians are at increased innate susceptibility or whether environmental factors related to migration and nutrition transitions are more at play. While South Asians have lower rates of obesity compared with other ethnic groups, as defined by BMI, they tend to have larger waist measurements and waist to hip ratios, and therefore a greater degree of central obesity.

This is seen at any given level of BMI and has been associated with higher plasma insulin levels, increased insulin resistance, and a higher prevalence of T2DM. Again, despite similar total body fat content, South Asians had greater abdominal adiposity than did Caucasians.

A cross-sectional study examining body size, body composition, and fat distribution among European, Maori, Pacific Island, and South Asian adults reported that South Asians had more total fat, both overall and in the abdominal region, compared with Europeans and Pacific Islanders. Such differences in body fat composition are seen even in childhood and adolescence.

Cross-sectional studies have shown that South Asian children have significantly more body fat than European children, with a greater degree of central fat. Prospective studies from India have shown that participants who were underweight as children had a high prevalence of overweight, obesity, and central obesity as young adults.

Furthermore, exposure to excess caloric energy later in life can lead to metabolic imbalances, thus increasing the risk for T2DM in adulthood, as several studies have noted that participants who were small at birth and became obese in childhood have and increased risk of developing insulin resistance. Several biomarkers, such as leptin, adiponectin, C-reactive protein CRP , and markers of oxidative stress may indicate processes that may influence the increased prevalence of T2DM in South Asians compared to other ethnic groups.

Oxidative stress resulting from an increased content of reactive oxygen species ROS has been implicated in the parthenogenesis of T2DM development.

Adiponectin and leptin are proteins linked with T2DM development. Circulating adiponectin concentrations are decreased in those with T2DM, while leptin levels increase with increasing BMI. In recent decades important demographic and environmental shifts have occurred in South Asia. Life expectancy is increasing while birth rates are declining, resulting in a significant proportion of older individuals.

It is well established that reductions in physical activity increase the risk of T2DM in all ethnic groups. In addition to lower physical activity levels, higher intakes of refined carbohydrates and saturated and trans fats have been shown to increase T2DM risk by adversely affecting glucose metabolism and insulin resistance.

In contrast, low glycemic index foods and foods high in dietary fiber have been shown to reduce glycemic and insulinemic responses, thereby reducing the risk of T2DM. Several micronutrients such as magnesium, calcium, vitamin C, and folate have been thought to play a beneficial role in glucose homeostasis.

Several studies have established the association between cigarette smoking and increased risk for T2DM. Duration of sleep, both above and below average, has been shown to play a role in T2DM risk.

Studies have noted that sleep restriction to only four hours caused decreases in the appetite suppressing hormone leptin and increases in the appetite inducing hormone ghrelin. Sleep duration of less than seven hours was a significant predictor of T2DM in Caucasians and Hispanics, even after adjusting for insulin sensitivity and insulin resistance.

A prospective, population based study from Sweden followed nondiabetic participants for approximately 15 years. Study results indicate that difficulties falling asleep or regular use of hypnotics were associated with an increased risk of developing diabetes.

Furthermore, a study examining the associations between sleep apnea and risk factors for metabolic syndrome in North India suggested that obstructive sleep apnea was independently positively associated with fasting insulin levels.

Persistent organic pollutants POPs encompass a variety of man-made chemicals and are of recent concern with regard to T2DM risk.

National Health and Examination Survey data reported strong associations between insulin resistance and serum concentrations of POPs.

However, the association between obesity and diabetes was strengthened as POP levels in the blood increased. To our knowledge, there have been no studies examining the relationship between POP exposure and diabetes risk in South Asian populations. However, there have been studies noting detectable levels of POP in mussels collected from the coastal waters of India. Current evidence suggests that the prevalence of T2DM in South Asians is high and rising both in South Asian countries, as well as in the diaspora.

These increases are due, in part, to higher T2DM incidence rates in South Asians compared with Caucasians, which suggests an increased propensity for South Asians to develop the disease.

In addition to possible innate predisposition, South Asians are currently experiencing changes in lifestyle behaviors due to migration or nutritional transitions, resulting in physical inactivity and a shift away from traditional dietary habits to those that include greater overall carbohydrates, saturated, and trans fats and lower amounts of dietary fiber.

Coupled with an increased propensity for T2DM, the recent shifts in lifestyle behaviors only serve to exacerbate the risk for disease. Future research is needed regarding the etiology and pathophysiology of disease in South Asians, compared with other ethnic groups.

Studies focusing on the contributions of tobacco use, sleep duration, and environmental pollutants are also warranted. Furthermore, there is a great need for primary prevention. This need will persist, especially as South Asians continue to become more affluent, have greater access to high-fat foods, adopt more sedentary lifestyles, experience a growing population of aging individuals, and migrate to diaspora countries.

Evidence indicates that lifestyle interventions, including increases in physical activity and improvements in dietary quality, are effective at preventing or delaying the development of T2DM in high-risk groups. Randomized controlled trials are now taking place to assess the effectiveness, cost-effectiveness, generalizability, and sustainability of such interventions in South Asians.

We thank Jessica Marcinkevage for her thoughtful review of the manuscript. National Center for Biotechnology Information , U.

Annals of the New York Academy of Sciences. Ann N Y Acad Sci. Published online Jan Author information Copyright and License information Disclaimer. Address for correspondence: Unjali P.

Gujral, M. Re-use of this article is permitted in accordance with the Creative Commons Deed, Attribution 2. This article has been cited by other articles in PMC. Abstract Type 2 diabetes mellitus T2DM is one of the leading causes of morbidity and mortality. Keywords: type 2 diabetes mellitus, South Asians, prevalence, ethnic comparison.

Introduction Type 2 diabetes mellitus T2DM currently affects approximately million people worldwide. Open in a separate window. Figure 1. Projected number of people with diabetes millions by year— and Figure 2. Differences in prevalence among South Asians and other ethnic groups. Table 2 Comparison of diabetes risk factors among South Asians, Caucasians, and other ethnic groups.

However, South Asians are shown to be shown to be more insulin resistant than Caucasian populations even at younger ages and lower levels of BMI. Some of this increased propensity for insulin resistance in South Asians might be attributed to greater deposition of visceral fat in South Asians as compared to Caucasians. While the actual genes established may not be different, some differences exist in allele frequencies and polymorphisms.

Mean age at diagnosis was lowest in South Asians 49 years , followed by Chinese 55 years , Blacks 57 years , and Whites 58 years living in Canada. However, despite similar levels of BMI, South Asians have more total abdominal and visceral fat compared to Caucasians.

Age It is a widely held belief that T2DM onset occurs at a much earlier age in South Asians than in other ethnic populations. Anthropometry While South Asians have lower rates of obesity compared with other ethnic groups, as defined by BMI, they tend to have larger waist measurements and waist to hip ratios, and therefore a greater degree of central obesity. Biomarkers Several biomarkers, such as leptin, adiponectin, C-reactive protein CRP , and markers of oxidative stress may indicate processes that may influence the increased prevalence of T2DM in South Asians compared to other ethnic groups.

Environmental risk factors for T2DM Lifestyle behaviors In recent decades important demographic and environmental shifts have occurred in South Asia.

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